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Dopamine-induced Tyrosine Phosphorylation of NR2B (Tyr1472) is Essential for ERK1/2 Activation and Processing of Novel Taste Information. Macromolecular complexes at active zones: integrated nano-machineries for neurotransmitter release. Spontaneous behavior in the social homecage discriminates strains, lesions and mutations in mice. Tomosyn-2 is required for normal motor performance in mice and sustains neurotransmission at motor endplates. Currently there are no events.

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Dopamine-induced Tyrosine Phosphorylation of NR2B (Tyr1472) is Essential for ERK1/2 Activation and Processing of Novel Taste Information. Macromolecular complexes at active zones: integrated nano-machineries for neurotransmitter release. Spontaneous behavior in the social homecage discriminates strains, lesions and mutations in mice. Tomosyn-2 is required for normal motor performance in mice and sustains neurotransmission at motor endplates. Currently there are no events.
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Dopamine-induced Tyrosine Phosphorylation of NR2B (Tyr1472) is Essential for ERK1/2 Activation and Processing of Novel Taste Information. Macromolecular complexes at active zones: integrated nano-machineries for neurotransmitter release. Spontaneous behavior in the social homecage discriminates strains, lesions and mutations in mice. Tomosyn-2 is required for normal motor performance in mice and sustains neurotransmission at motor endplates. Currently there are no events.

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Dopamine-induced Tyrosine Phosphorylation of NR2B (Tyr1472) is Essential for ERK1/2 Activation and Processing of Novel Taste Information. David O, Barrera I, Adaikkan C, Kaphzan H, Nakazawa T, Yamamoto T, Rosenblum K. Front Mol. Neurosci. doi: 10.3389/fnmol.2014.00066 [Epub ahead of print]. Read more Dopamine-induced Tyrosine Phosphorylation of NR2B (Tyr1472) is Essential for ERK1/2 Activation and Processing of Novel Taste Information. Cell Mol Life Sci. 2014 Jun 10. [Epub ahead of print]. A solution to ...

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Spontaneous behavior in the social homecage discriminates strains, lesions and mutations in mice. - EUROSPIN

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Spontaneous behavior in the social homecage discriminates strains, lesions and mutations in mice. Vannoni E, Voikar V, Colacicco G, Sánchez MA, Lipp HP, Wolfer DP. J Neurosci Methods. 2014 May 1. pii: S0165-0270(14)00142-3. doi: 10.1016/j.jneumeth.2014.04.026. We asked whether the analysis of spontaneous. During the first week in the social. Tested in the IntelliCage during the past years. We then computed individual component scores to characterize and compare groups of mice. We found 11 uncorrelated co...

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Dopamine-induced Tyrosine Phosphorylation of NR2B (Tyr1472) is Essential for ERK1/2 Activation and Processing of Novel Taste Information. Macromolecular complexes at active zones: integrated nano-machineries for neurotransmitter release. Spontaneous behavior in the social homecage discriminates strains, lesions and mutations in mice. Tomosyn-2 is required for normal motor performance in mice and sustains neurotransmission at motor endplates. Currently there are no events.

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Start - EUROSPIN

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In het project participeren 15 wetenschappelijke groepen en drie ondernemingen. Het project ontvangt een bijdrage van maximaal 11.9 miljoen euro van de Europese Commissie voor een periode van vier jaar. Op deze website vindt u (in het Engelstalige deel) informatie over de EUROSPIN-groepen en ons wetenschappelijk concept. De website zal regelmatig worden geactualiseerd en uitgebreid met resultaten, publicaties, interactieve databases enz. Voor vragen kunt u zich wenden tot de projectmanager.

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Tomosyn-2 is required for normal motor performance in mice and sustains neurotransmission at motor endplates. - EUROSPIN

http://eurospin.mpg.de/publications-reader/items/tomosyn-2-is-required-for-normal-motor-performance-in-mice-and-sustains-neurotransmission-at-motor-endplates.html

Tomosyn-2 is required for normal motor performance in mice and sustains neurotransmission at motor endplates. Geerts CJ, Plomp JJ, Koopmans B, Loos M, van der Pijl EM, van der Valk MA, Verhage M, Groffen AJ. Brain Struct Funct. 2014 Apr 18. [Epub ahead of print]. Neuronal exocytosis is mediated by soluble N-ethylmaleimide-sensitive factor attachment protein. Proteins. Before fusion. Proteins form complexes bridging the membrane. Followed by assembly toward the C-terminal membrane.

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CNCR | Clinical Neurosciences

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Animal Models of Psychiatric Disease. Glial Regulated Neuronal Plasticity. Secretory vesicle trafficking and recycling. Molecular Mechanisms of Exocytosis. Neuronal Oscillations and Cognition. Phenotypic modelling in genetics. Vesicle Dynamics and Synaptic Plasticity. Mechanisms of Brain Disorders. Department of Functional Genomics. Department of Integrative Neurophysiology. Department of Complex Trait Genetics. Department of Molecular and Cellular Neurobiology. Word from the director.

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CNCR | Behavior

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Animal Models of Psychiatric Disease. Glial Regulated Neuronal Plasticity. Secretory vesicle trafficking and recycling. Molecular Mechanisms of Exocytosis. Neuronal Oscillations and Cognition. Phenotypic modelling in genetics. Vesicle Dynamics and Synaptic Plasticity. Mechanisms of Brain Disorders. Department of Functional Genomics. Department of Integrative Neurophysiology. Department of Complex Trait Genetics. Department of Molecular and Cellular Neurobiology. Word from the director.

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CNCR | Department of Complex Trait Genetics

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Animal Models of Psychiatric Disease. Glial Regulated Neuronal Plasticity. Secretory vesicle trafficking and recycling. Molecular Mechanisms of Exocytosis. Neuronal Oscillations and Cognition. Phenotypic modelling in genetics. Vesicle Dynamics and Synaptic Plasticity. Mechanisms of Brain Disorders. Department of Functional Genomics. Department of Integrative Neurophysiology. Department of Complex Trait Genetics. Department of Molecular and Cellular Neurobiology. Word from the director. Research in our lab.

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CNCR | Department of Functional Genomics

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Animal Models of Psychiatric Disease. Glial Regulated Neuronal Plasticity. Secretory vesicle trafficking and recycling. Molecular Mechanisms of Exocytosis. Neuronal Oscillations and Cognition. Phenotypic modelling in genetics. Vesicle Dynamics and Synaptic Plasticity. Mechanisms of Brain Disorders. Department of Functional Genomics. Department of Integrative Neurophysiology. Department of Complex Trait Genetics. Department of Molecular and Cellular Neurobiology. Word from the director. In our studies of ...

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CNCR | Themes

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Animal Models of Psychiatric Disease. Glial Regulated Neuronal Plasticity. Secretory vesicle trafficking and recycling. Molecular Mechanisms of Exocytosis. Neuronal Oscillations and Cognition. Phenotypic modelling in genetics. Vesicle Dynamics and Synaptic Plasticity. Mechanisms of Brain Disorders. Department of Functional Genomics. Department of Integrative Neurophysiology. Department of Complex Trait Genetics. Department of Molecular and Cellular Neurobiology. Word from the director.

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CNCR | Expertise

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Animal Models of Psychiatric Disease. Glial Regulated Neuronal Plasticity. Secretory vesicle trafficking and recycling. Molecular Mechanisms of Exocytosis. Neuronal Oscillations and Cognition. Phenotypic modelling in genetics. Vesicle Dynamics and Synaptic Plasticity. Mechanisms of Brain Disorders. Department of Functional Genomics. Department of Integrative Neurophysiology. Department of Complex Trait Genetics. Department of Molecular and Cellular Neurobiology. Word from the director.

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CNCR | Proteomics

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Animal Models of Psychiatric Disease. Glial Regulated Neuronal Plasticity. Secretory vesicle trafficking and recycling. Molecular Mechanisms of Exocytosis. Neuronal Oscillations and Cognition. Phenotypic modelling in genetics. Vesicle Dynamics and Synaptic Plasticity. Mechanisms of Brain Disorders. Department of Functional Genomics. Department of Integrative Neurophysiology. Department of Complex Trait Genetics. Department of Molecular and Cellular Neurobiology. Word from the director. Proteomics experim...

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CNCR | Mechanisms of Brain Disorders

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Animal Models of Psychiatric Disease. Glial Regulated Neuronal Plasticity. Secretory vesicle trafficking and recycling. Molecular Mechanisms of Exocytosis. Neuronal Oscillations and Cognition. Phenotypic modelling in genetics. Vesicle Dynamics and Synaptic Plasticity. Mechanisms of Brain Disorders. Department of Functional Genomics. Department of Integrative Neurophysiology. Department of Complex Trait Genetics. Department of Molecular and Cellular Neurobiology. Word from the director.

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Dal 19 al 31 marzo 2018. 25 anni di Spesa Intelligente. Dall’8 al 31 marzo 2018. Promozione valida dal 1 al 31 marzo. Scopri i ribassati di Marzo. Le occasioni della settimana. Dai migliori accessori per la casa, a quelli dell’auto o al tessile: scopri quali sono le nostre selezioni di questa settimana! Eurospin ti offre un'ampia gamma di prodotti rigorosamente di proprietà o concessi in esclusiva da fornitori selezionati con scrupolosità. La linea di prodotti per intolleranti a lattosio e glutine. Trova...

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Home - EUROSPIN

Dopamine-induced Tyrosine Phosphorylation of NR2B (Tyr1472) is Essential for ERK1/2 Activation and Processing of Novel Taste Information. Macromolecular complexes at active zones: integrated nano-machineries for neurotransmitter release. Spontaneous behavior in the social homecage discriminates strains, lesions and mutations in mice. Tomosyn-2 is required for normal motor performance in mice and sustains neurotransmission at motor endplates. Currently there are no events.

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Home page - EUROSPINdb database of the EUROSPIN project

Is the central integrating project database of the EUROSPIN project. With the dual aim of collecting all project data at a single place allowing the project members to easily search relevant results. In a second step towards the end of the project, structured data will be made open to the scientific community to participate in discovering fundamental biological principles and important insights into brain disease. Are now available from the EUROSPIN project. This site is hosted by Edinburgh University.

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Neurosurgical Spine Center Hannover. Prof Piepgras and Partners. We are specialised in minimally invasive, microsurgical procedures of the spine, e.g herniated, ruptured discs or degenerative disease of the spine ( stenosis ). In-hospital stay is reduced to a few days, therefore. We offer fast scheduling with minimal waiting time for surgery, usually within a few days. Recommended and supported by the leading german insurance companies. Contact via email preferred: info@eurospine.de.

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