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High-density lipoprotein dysfunction in development of cardiovascular disease and as a therapeutic target. One explanation for the lack of effect is that the biologic activities of HDL, rather than simply its level in the blood, contribute to its beneficial effect. Indeed, there is evidence that HDL becomes dysfunctional and loses its protective properties in patients with atherosclerosis. Transatlantic research network supported by a grant from the Fondation Leducq.

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Home | HDL | hdl-network-leducq.com Reviews

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High-density lipoprotein dysfunction in development of cardiovascular disease and as a therapeutic target. One explanation for the lack of effect is that the biologic activities of HDL, rather than simply its level in the blood, contribute to its beneficial effect. Indeed, there is evidence that HDL becomes dysfunctional and loses its protective properties in patients with atherosclerosis. Transatlantic research network supported by a grant from the Fondation Leducq.

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3 publications

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5 news

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7 high density lipoprotein cholesterol

8 coupons

9 reviews

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Home | HDL | hdl-network-leducq.com Reviews

https://hdl-network-leducq.com

High-density lipoprotein dysfunction in development of cardiovascular disease and as a therapeutic target. One explanation for the lack of effect is that the biologic activities of HDL, rather than simply its level in the blood, contribute to its beneficial effect. Indeed, there is evidence that HDL becomes dysfunctional and loses its protective properties in patients with atherosclerosis. Transatlantic research network supported by a grant from the Fondation Leducq.

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hdl-network-leducq.com

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Network Map | HDL

http://www.hdl-network-leducq.com/networkmap

High-density lipoprotein dysfunction in development of cardiovascular disease and as a therapeutic target. Alan M. Fogelman, M.D. Is Castera Professor, Director of the Atherosclerosis Research Unit and Executive Chair of the Department of Medicine at the David Geffen School of Medicine at UCLA. His research focuses on the mechanisms by which HDL modulates inflammation and the use of apolipoprotein mimetic pep-tides to reduce chronic inflammation in atherosclerosis and vascular dementia. He worked on plas...

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No actual news | HDL

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High-density lipoprotein dysfunction in development of cardiovascular disease and as a therapeutic target. There are no news available at the moment. Transatlantic research network supported by a grant from the Fondation Leducq.

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Thomas F. Lüscher, M.D. | HDL

http://www.hdl-network-leducq.com/members/thomas-f-lüscher-md

High-density lipoprotein dysfunction in development of cardiovascular disease and as a therapeutic target. Thomas F. Lüscher, M.D. Alan M. Fogelman, M.D. John E. Deanfield, M.D. Stanley Hazen, MD, Ph.D. Jan Albert Kuivenhoven Ph.D. Alan Tall, M.D., Ph.D. Bart Staels, Ph.D. Ulf Landmesser, M.D. Thomas F. Lüscher, M.D. Is Professor and Chairman of Cardiology and CardioVascular Research at the University Zurich, Switzerland. Transatlantic research network supported by a grant from the Fondation Leducq.

4

Links | HDL

http://www.hdl-network-leducq.com/links

High-density lipoprotein dysfunction in development of cardiovascular disease and as a therapeutic target. Zurich Heart House - Foundaton for Cardiovascular Research. Transatlantic research network supported by a grant from the Fondation Leducq.

5

Network related | HDL

http://www.hdl-network-leducq.com/content/network-related

High-density lipoprotein dysfunction in development of cardiovascular disease and as a therapeutic target. Chattopadhyay A, et al., & Fogelman AM. A novel approach to oral apoA-I mimetic therapy. J Lipid Res. 2013;54(4):995-1010. Ganda A, et al., & Tall AR. Mild renal dysfunction and metabolites tied to low HDL cholesterol are associated with monocytosis and atherosclerosis. Circulation. 2013;127(9):988-96. Gogonea V, et al., & Hazen SL. J Lipid Res. 2013;54(4):966-83. Holleboom AG, ., Kuivenhoven JA.

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Home | HDL

High-density lipoprotein dysfunction in development of cardiovascular disease and as a therapeutic target. One explanation for the lack of effect is that the biologic activities of HDL, rather than simply its level in the blood, contribute to its beneficial effect. Indeed, there is evidence that HDL becomes dysfunctional and loses its protective properties in patients with atherosclerosis. Transatlantic research network supported by a grant from the Fondation Leducq.

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