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Muthuswamy Laboratory

Human breast tissue immunostained for the cell-cell junction protein, E-cadherin (Green); the apical membrane protein, Muc-1 (Pink); the tight junction protein, ZO-1 (Red) and DNA (blue). It is likely a deeper understanding of oncogenes that disrupt cell and tissue architecture will identify new targets for controlling early lesions. Our current efforts are focused on: 1) How do oncogenes target cell polarity pathways? 4) Are pathways that control cell polarity novel targets for chemoprevention?

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Human breast tissue immunostained for the cell-cell junction protein, E-cadherin (Green); the apical membrane protein, Muc-1 (Pink); the tight junction protein, ZO-1 (Red) and DNA (blue). It is likely a deeper understanding of oncogenes that disrupt cell and tissue architecture will identify new targets for controlling early lesions. Our current efforts are focused on: 1) How do oncogenes target cell polarity pathways? 4) Are pathways that control cell polarity novel targets for chemoprevention?
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Muthuswamy Laboratory | muthuswamylab.cshl.edu Reviews

https://muthuswamylab.cshl.edu

Human breast tissue immunostained for the cell-cell junction protein, E-cadherin (Green); the apical membrane protein, Muc-1 (Pink); the tight junction protein, ZO-1 (Red) and DNA (blue). It is likely a deeper understanding of oncogenes that disrupt cell and tissue architecture will identify new targets for controlling early lesions. Our current efforts are focused on: 1) How do oncogenes target cell polarity pathways? 4) Are pathways that control cell polarity novel targets for chemoprevention?

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Muthuswamy Laboratory

Human breast tissue immunostained for the cell-cell junction protein, E-cadherin (Green); the apical membrane protein, Muc-1 (Pink); the tight junction protein, ZO-1 (Red) and DNA (blue). It is likely a deeper understanding of oncogenes that disrupt cell and tissue architecture will identify new targets for controlling early lesions. Our current efforts are focused on: 1) How do oncogenes target cell polarity pathways? 4) Are pathways that control cell polarity novel targets for chemoprevention?

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